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Cell子刊:北京大学强力/汪丽恒团队揭示肥胖中IgG积累驱动胰岛素 ...
在最新研究中,研究团队进一步确定了IgG在肥胖发生中特异性堆积在脂肪组织中,这种积累是由新生儿Fc受体(FcRn)依赖的再循环控制的,分别在饮食诱导的肥胖(DIO)的早期和晚期阶段,在脂肪祖细胞和巨噬细胞中调控。靶向FcRn可以抑制IgG的积累,纠正 ...
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